We ’ve madehuge advances toward conquering HIV , the immune - system - disabling computer virus that causesAIDS . But this disease still subsist , cause nearly two million end per year , in declamatory part because the computer virus recrudesce drug - resistant mutations so quick . investigator at Drexel University have a different access — a molecule that play tricks the computer virus into self - destructing before it infects cells .
https://gizmodo.com/were-closer-than-you-think-to-beating-hiv-700359471
Like all viruses , HIV work by latching onto healthy cells and stick in its gene , turning the cells into factories that fill up with HIV and burst . Drexel University ’s Dual Action Virolytic Entry Inhibitor , or DAVEI , combines a modified component of HIV ’s cell - bind mechanism with a protein that tricks the computer virus into open up its protective software . DAVEI selectively binds HIV and create the virus respond as if it ’s seize to a cell , spitting out its content . By break up the ingredient of the virus and destroying the space capsule needed to insert them into a prison cell , DAVEI supply the virus harmless .
Since far-flung HIV infection in a patient ’s immune organization is what causes AIDS , destroy the virus cursorily could keep the immunodeficiency from ever take postponement . Further research is take , of form , but a targeted mechanism that mechanically inactivates HIV could help fight down even drug - resistant viral strains . [ MedicalXpress ]
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